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The Simini Boards-Cast is the go-to audio study tool for small animal surgery residents prepping for board exams.
Each episode simplifies high-yield surgical content from trusted sources — built to help you pass faster and with less stress.
🎧 Audio-based learning for passive study
✂️ Practical relevance for surgical application
🧠 Flashcard-style recaps + board-style questions
📈 Designed with resident + program director input
Whether you're commuting, walking the dog, or post-op, turn that time into surgical mastery.
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143 Episodes
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In this BoardsCast episode, we finish Tobias Chapter 101 — Larynx with the harsh reality of laryngeal paralysis: You can’t fix the nerve; you can only fix the airway. Arytenoid lateralization (“tieback”) is not “restoring function.” It’s a permanent mechanical redesign of the airway gate — forcing the larynx open so the patient can breathe, even though the biologic motor is permanently offline. You’ll learn: The dominant model: tieback = mechanical redesign (not nerve recovery) Why...
In this BoardsCast episode, we continue Tobias Chapter 101 — Larynx by deleting the most common diagnostic mistake in upper airway medicine: You cannot diagnose the airway by listening to it. Stridor is an alarm, not a diagnosis. The sound tells you there’s a restriction somewhere, but it cannot tell you what is mechanically failing. This episode rebuilds laryngeal evaluation into the only board-safe framework: Laryngeal disease is diagnosed by watching the gate move. You’ll learn: Why noise ...
In this BoardsCast episode, we continue Tobias Chapter 101 — Larynx with one of the most terrifying presentations in small animal practice: The lungs are fine, the airway isn’t blocked, and the dog still can’t breathe. That’s because laryngeal paralysis isn’t a lung disease, it’s a mechanical failure of the airway gate. If you remember one model from this episode, make it this: Laryngeal paralysis = failure of the airway gate to open during inspiration. You’ll learn: Why does normal breathing...
In this BoardsCast episode, we continue Tobias Chapter 101 — Larynx by dismantling the most dangerous misconception in respiratory mechanics: The airway is not a passive tube, It’s a spring-loaded gate that must be actively pulled open with every breath. This episode rebuilds the entire “how breathing works” model using one visual you won’t forget: the larynx is a set of heavy double doors — and one muscle is the only motor that opens them. You’ll learn: Why inspiration requires active ...
In this BoardsCast episode, we begin Tobias Chapter 101 — Larynx with the scary clinical paradox: the nose is clear, the trachea is open, but the dog still can’t breathe. That’s because the problem isn’t the “tubing, It’s the gate in the middle — the larynx. This episode rebuilds the mental model the boards want you to have cold: The larynx is a dynamic airway valve with three jobs — and it must switch between them flawlessly: airflow regulation, airway protection, and phonation. You’ll learn...
In this BoardsCast episode, we finish Tobias Chapter 100 - Palate by reframing brachycephalic airway syndrome with the only honest model: This isn’t “noisy breathing.” It’s an animal forcefully pulling air against a physical wall. The dominant framework is simple: Brachycephalic syndrome is a packaging error. The skull got shorter — the soft tissue didn’t. And the primary internal culprit is the overlong soft palate, which physically invades the laryngeal opening and starts a cascade o...
In this BoardsCast episode, we continue Tobias Chapter 100 - Palate with the ultimate palatal nightmare: You close the defect perfectly, and the sutures look perfect. Two weeks later, the hole is back. This episode delivers the reframe that fixes most palatal failures: When a palatal repair fails, it’s not a suturing problem. It’s a blood supply + tension problem. You’ll learn: The 2 non-negotiables: preserve vascular supply and eliminate tensionWhy the major palatine artery is the hard...
In this BoardsCast episode, we continue Tobias Chapter 100 - Palate by reframing palatal disease with the only mental model that matters: This is not a feeding problem.It’s a barrier failure. When the palate fails, the oral cavity and nasal passages stop being separate systems. Food, liquid, and oral bacteria cross into the nose — and the downstream consequence is predictable: chronic rhinitis, nasal reflux, and aspiration risk. You’ll learn: The separation system: hard palate = wall, s...
In this BoardsCast episode, we continue Tobias Chapter 100 - Palate with the mental model that makes soft palate pathology finally click: The soft palate is not a flap. It’s a valve. Every swallow is a routing decision. Air and food can’t share the same lane — so the soft palate runs a dynamic “switching system” that separates breathing from swallowing, in real time. You’ll learn: The core model: soft palate = moving valve (not passive tissue) The 4-muscle “engine” that runs the va...
In this BoardsCast episode, we begin Tobias Chapter 100 - Palate by reframing the palate the way the boards want you to think: The palate isn’t the roof of the mouth. It’s a separation mechanism that lets breathing and eating happen at the same time. The palate’s job only becomes obvious when it fails. When the barrier is breached (cleft, trauma, or dehiscence), breathing and eating collide — food and liquid reflux into the nose, contamination becomes chronic, and the endgame risk is as...
In this BoardsCast episode, we finish Tobias Chapter 99 — Nasal Planum, Nasal Cavity, and Sinuses by reframing the most stressful moment in nasal disease: When surgery enters the nose, you’re not fighting the incision — you’re fighting the chaos inside. Nasal surgery is notorious for three unavoidable realities: extreme vascularity, complex anatomy in a tiny space, and predictable post-op complications. If you don’t plan for all three, the case fails before you touch the lesion. You’ll learn:...
In this BoardsCast episode, we continue Tobias Chapter 99 — Nasal Planum, Nasal Cavity, and Sinuses by solving the #1 reason chronic nasal cases drag on forever: People treat the symptom instead of the category. Tobias Chapter 99 makes the framework brutally simple: most chronic nasal disease lives in three buckets — and your job is to sort the patient into the right bucket before you chase a microscopic diagnosis. Tumor. Fungus. Inflammation. You’ll learn: The core rule: categorize first, di...
In this BoardsCast episode, we continue Tobias Chapter 99 — Nasal Planum, Nasal Cavity, and Sinuses with the most important diagnostic framework in upper airway medicine: Sneezing isn’t a diagnosis. It’s an alarm. This episode builds the board-safe model that stops symptom-chasing and starts winning cases: Nasal disease is solved by localization first, diagnosis second. You’ll learn: Why nasal signs are “shared alarms” across wildly different diseasesThe 3 regions to localize: nasal cavity / ...
In this BoardsCast episode, we continue Tobias Chapter 99 — Nasal Planum, Nasal Cavity, and Sinuses with the core reframe that changes how nasal disease makes sense: The nose is not just an airway. It conditions, monitors, and protects every breath before it reaches the lungs. If you bypass or destroy nasal function (intubation, obstruction, turbinate loss), you don’t just change airflow — you change the entire respiratory landscape. The lower airways inherit raw environmental air, whic...
In this BoardsCast episode, we begin Tobias Chapter 99 — Nasal Planum, Nasal Cavity, and Sinuses by fixing the “nasal cases are a black box” problem with one mental model: The nose is not a tube. It’s a maze. Once you stop thinking “hollow pipe,” nasal signs stop feeling random. Disease patterns follow anatomy, and the clinical signs tell you where the maze is blocked. You’ll learn: The airflow map: nares → cavity → choanaeWhy turbinates matter (they create the maze)The “four lanes”: ventral ...
In this BoardsCast episode, we finish Tobias Chapter 98 with the scenario every clinician has lived: The tube came out… and two days later, the patient stopped eating. This episode reframes tube removal the way the boards (and real life) demand: removing a feeding tube is not the end of a treatment plan. It’s a calculated transition decision—and most failures happen because the tube is abandoned too soon. The dominant mental model is simple: A feeding tube is a bridge to physiologic in...
In this BoardsCast episode, we continue Tobias Chapter 98 with one of the most terrifying clinical reversals: The feeding worked… and the patient crashed. This episode rebuilds the mental model that prevents that disaster: Enteral nutrition is not calorie delivery — it’s a metabolic restart. A starved patient is running on a low-insulin, catabolic “idle.” When you suddenly deliver a full carbohydrate load, you trigger a massive insulin surge — and that surge drives electrolytes out of the blo...
In this BoardsCast episode, we continue Tobias Chapter 98 by confronting the most common feeding-tube reality: The tube was perfect. The complications weren’t. Feeding tubes rarely fail during placement. They fail during management — because once a tube is placed, you’ve created a new portal into the GI tract, a new wound, and a foreign body that lives or dies based on daily handling. This episode builds the practical framework the boards want you to know: most “tube disasters” are pred...
In this BoardsCast episode, we continue Tobias Chapter 98 by confronting a uniquely frustrating ICU moment: The tube worked… and the patient still declined. Because feeding tubes are not procedural decisions. They’re physiologic access decisions—and the route you choose changes the biology of how the patient tolerates, digests, and absorbs nutrition. The dominant mental model for this episode is simple: The feeding route determines the physiologic response. You’ll learn: Why GI segment ...
In this BoardsCast episode, we begin Tobias Chapter 98 by dismantling the most expensive mistake in postoperative care: The surgery went perfectly… and the patient still declined. That outcome usually isn’t hardware failure or incision failure. It’s a metabolic failure because malnutrition drives surgical failure, even when the operation succeeds. This episode rebuilds your mental model around one dominant truth: Nutrition is active metabolic treatment — not “supportive care.” You’ll le...
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